Compendium
Cognitive

N-Acetyl Selank Amidate

An enhanced, stabilized form of Selank with increased receptor binding affinity. It modulates GABA-A receptors and serotonin/dopamine systems for anxiolysis and cognitive enhancement.

Protocol

100–300 mcg intranasal, 1–2x daily

Half-Life

1–2 hours

Molecular Structure & Chemistry

20 residues • 346 atoms • Skeletal Formula

Formula

C122H168N30O25S

Molar Mass

2,126.64 g/mol

Exact Mass

2485.252 Da

Isoelectric Pt

9.77

Charge pH 7

+1.94

GRAVY

+0.11

Hover Cα carbons for residue info

Amino Acid Sequence

KFWVPAWEGYCRSLAWAFLR

Biological Mechanisms

Potent anxiolytic

Observed in clinical research as a primary mechanism of action for N-Acetyl Selank Amidate.

Enhanced cognition

Observed in clinical research as a primary mechanism of action for N-Acetyl Selank Amidate.

Memory improvement

Observed in clinical research as a primary mechanism of action for N-Acetyl Selank Amidate.

Mood stabilization

Observed in clinical research as a primary mechanism of action for N-Acetyl Selank Amidate.

Immune modulation

Observed in clinical research as a primary mechanism of action for N-Acetyl Selank Amidate.

Safety & Risk Analysis

Nasal irritationMild sedationRare headache

Critical Safety Warning

Black market peptides can be contaminated, mislabeled, or dangerous. Only obtain peptides via a registered doctor and legitimate pharmacy. Never attempt self-administration without professional medical supervision.

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Quick Reference

CategoryCognitive
Half-Life1–2 hours
AdminSubcutaneous
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Medical Note

This compound is for research purposes only. Clinical applications must be directed by a licensed physician.

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