Compendium
Cognitive

N-Acetyl Semax Amidate

N-Acetyl Semax Amidate has N-acetylation and C-terminal amidation that dramatically increase potency and duration of effect compared to standard Semax.

Protocol

100–300 mcg intranasal, 1–2x daily

Half-Life

Minutes to hours

Molecular Structure & Chemistry

20 residues • 314 atoms • Skeletal Formula

Formula

C111H149N27O26S

Molar Mass

1,949.33 g/mol

Exact Mass

2308.090 Da

Isoelectric Pt

8.79

Charge pH 7

+1.03

GRAVY

-0.4

Hover Cα carbons for residue info

Amino Acid Sequence

KFWVPAWEGYCGSHFGPVKN

Biological Mechanisms

Enhanced cognitive performance

Observed in clinical research as a primary mechanism of action for N-Acetyl Semax Amidate.

Greater BDNF upregulation

Observed in clinical research as a primary mechanism of action for N-Acetyl Semax Amidate.

Reduced anxiety

Observed in clinical research as a primary mechanism of action for N-Acetyl Semax Amidate.

Improved focus

Observed in clinical research as a primary mechanism of action for N-Acetyl Semax Amidate.

Longer-lasting effects

Observed in clinical research as a primary mechanism of action for N-Acetyl Semax Amidate.

Safety & Risk Analysis

Irritability at high dosesNasal irritationOverstimulationInsomnia

Critical Safety Warning

Black market peptides can be contaminated, mislabeled, or dangerous. Only obtain peptides via a registered doctor and legitimate pharmacy. Never attempt self-administration without professional medical supervision.

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Quick Reference

CategoryCognitive
Half-LifeMinutes to hours
AdminSubcutaneous
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Medical Note

This compound is for research purposes only. Clinical applications must be directed by a licensed physician.

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